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Heart Failure

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Gender differences in heart failure: paving the way towards personalized medicine?

Eur. Heart J. (2010), 31 (10), 1165-1167; 10.1093/eurheartj/ehq073 - Click here to view abstract

Gender differences in heart failure are based on distinct characteristics in diagnosis, management, and prognosis in men and women. Molecular details of cardiac pathophysiology may influence the clinical phenotype. Genes differentially expressed (top) and phenotypic characteristics (bottom) are depicted relative to the other gender, where the left column represents genes and characteristics more prevalent in women, and the right column those more prevalent in men.

 

The Swedish paradox: or is there really no gender difference in acute coronary syndromes? - Figure 1

Eur. Heart J. (2011) 32 (24), 3070; 10.1093/eurheartj/ehr375 - Click here to view abstract

Difficulties in detection of acute coronary syndromes.

 

Novel insights on HIV/AIDS and cardiac disease: shedding light on the HAART of Darkness

Eur. Heart J. (2011) 33 (7), 813; 10.1093/eurheartj/ehr413 - Click here to view abstract

Disentangling the conundrum of aetiology, pathogenesis, and clinical manifestation of cardiac disease in human immunodeficiency virus/acquired immunodeficiency syndrome (HIV/AIDS). HAART, highly active antiretroviral therapy; IRIS, immune reconstitution inflammatory syndrome; RHF, right heart failure.

 

Iron deficiency and heart failure: diagnostic dilemmas and therapeutic perspectives - Figure 1

Eur Heart J (2013) 34 (11): 816-829; 10.1093/eurheartj/ehs224 - Click here to view abstract

Importance of iron for functioning and survival across all levels of complexity of living structures.

 

Iron deficiency and heart failure: diagnostic dilemmas and therapeutic perspectives - Figure 2

Eur Heart J (2013) 34 (11): 816-829; 10.1093/eurheartj/ehs224 - Click here to view abstract 

Major pools of utilized and stored iron in the body.

 

Iron deficiency and heart failure: diagnostic dilemmas and therapeutic perspectives - Figure 3

Eur Heart J (2013) 34 (11): 816-829; 10.1093/eurheartj/ehs224 - Click here to view abstract 

The concept of absolute and functional iron deficiency.

 

Iron deficiency and heart failure: diagnostic dilemmas and therapeutic perspectives - Figure 4

Eur Heart J (2013) 34 (11): 816-829; 10.1093/eurheartj/ehs224 - Click here to view abstract

Tissues utilizing and/or storing iron and related biomarkers which are secreted by these tissues and can be detected in peripheral blood. 

 

What does the liver tell us about the failing heart - Figure 1

Eur Heart J (2013) 34 (10): 711-714; 10.1093/eurheartj/ehs440 - Click here to view abstract 

Haemodynamic disturbances in heart failure and mechanisms resulting in different patterns of elevated liver enzymes. Congestive hepatic injury (left panel) and ischaemic hepatic injury (right panel).

 

Reversing heart failure by CRT: how long do the effects last? - Figure 1

Eur Heart J (2013) 34 (33): 2582-2584; 10.1093/eurheartj/eht238 - Click here to view abstract 

Long-term impact of different ventricular activation patterns on regional load and hypertrophy. A, delayed LV activation in LBBB unloads the septum and increases the regional load in the delayed activated postero-lateral wall resulting in compensatory hypertrophy.8 B, Optimized CRT may normalize these pathologic relationships by simultaneous and more rapid ventricular activation. C, Hypothetical (and probably exaggerated) result of suboptimal CRT with early LV activation (reverse dyssynchrony). The early activated posterolateral wall is exposed to a lower regional load and the late activated opposing wall (i.e. the septum) responds with regional hypertrophy.

 

Non-pharmacological modulation of the autonomic tone to treat heart failure

Eur Heart J (2014) 35 (2): 77-85; 10.1093/eurheartj/eht436 - Click here to view abstract 

Role of brain and kidney in activation of the renin–angiotensin–aldosterone system in hypertension, and heart failure.

 

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