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European Heart Journal 1991 12(Supplement B):66-69; doi:10.1093/eurheartj/12.suppl_B.66
Copyright © 1991 by the European Society of Cardiology.
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© 1991 The European Society of Cardiology

Left ventricular function in rheumatic mitral stenosis

W. H. Gaasch and E. D. Folland

Department of medicine (Cardiology), The Medical Center Massachusetts/Memorial Worcester, Massachusetts, U.S.A.

Correspondence: William H. Gaasch, MD, Chief of Cardiology, The Med Center/Memorial, 119 Belmont St, Worcester, MA 01605, U.S.A.

Haemodynamic factors contributing to clinical disability in patients with rheumatic mitral stenosis have been under discussion and investigation for decades. Prior to the development of left heart catheterization, a low cardiac output in the presence of little or no pulmonary hyperternion was taken as evidence for a myocardial ‘insufficiency’. With the use of left heart catheterization, it was possible to exclude the presence of cororary artery disease and to assess directly the size and function of the left ventricle. Such studies indicate a tendency toward low-normal left ventricular end-diastolic volumes and low-normal ejection fractiorns. Modest reductions in the ejection fraction may be due to: (1) a restriction or tethering of posterobasal myocardium by the scarred mitral apparatus, or (2) abnormal interventricular septal motion related to right ventricular overload and unequal filling of the two ventricles. These and other factors, such as limited LV disternibility and variable diastolic suction, may affect ventricular function in rheumatic mitral stenosis. Thus, left ventricular dysfunction can generally be explained without implicating a rheumatic myocardial factor.

Key Words: Left ventricular function • left ventricular volume • mitral stenosis • rheumatic heart disease


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