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European Heart Journal 1995 16(Supplement O):15-17; doi:10.1093/eurheartj/16.suppl_O.15
Copyright © 1995 by the European Society of Cardiology.
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© 1995 The European Society of Cardiology

Genetics of coxsackievirus B3 cardiovirulence

S. Tracy, Z. Tu, N. Chapman and G. Hufnagel*

Department of Pathology and Microbiology, University of Nebraska Medical Center Omaha, NE 68198, U.S.A
* Klinikum der Philipps-Universitaet Marburg, Abteilung fuer Innere Medizin-Kardiologie Marburg, Germany

Steven Tracy, Department of Pathology and Microbiology, University of Nebraska Medical Center, 600 South 42nd Street, Omaha, NE 68198, U.S.A.

The human enteroviruses, especially the coxsackie B viruses, have been established as aetiologic agents of human inflammatory heart disease, a condition which may lead to dilated cardiomyopathy and heart failure. It is clear from murine models of coxsackievirus B3-induced inflammatory heart disease that not all strains of the virus are cardiovindent (able to cause disease). Here, we present preliminary data mapping the site in a coxsackievirus B3 genome which determines a cardiovindent phenotype.

Key Words: Coxsackievirus B3 • enterovirus • inflammatory heart disease


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