Skip Navigation

European Heart Journal 1986 7(6):528-537;
Copyright © 1986 by the European Society of Cardiology.
This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by WEBER, H.
Right arrow Articles by RAHLF, G.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by WEBER, H.
Right arrow Articles by RAHLF, G.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© 1986 The European Society of Cardiology

Percutaneous intracardiac direct-current shocks in dogs: arrhythmogenic potential and pathological changes

H. WEBER, L. SCHMITZ, R. DISCHE and G. RAHLF

Department of Paediatric Cardiology and Department ofPathology, University of Goettingen Goettingen, F.R.G.

Received 31 May 1985; revised 21 January 1986; .

Address for correspondence: Helmut Weber, MD, Lauterbacher Muehle, D-8124 Seeshaupt, F.R.G.

Abstract

In 12 dogs, a total of 65 direct-current (DC) shocks of 100-300 J were delivered through a standard USCI6F tripolar electrode catheter to selected sites in the heart. Severe arrhythmias were more frequent after electric shocks of high energy to the ventricles and AV-nodal or His-bundle region than after comparable shocks to the left or right atria. There was a direct relationship between the strength of the electric shocks, and the extent and severity of the injury. Application of 300 J shocks led to massive necrosis and damage to all components of the myocardium including the walls of small blood vessels. However, perforation of the atrial and ventricular walls or septum did not occur in any of the animals (mean follow-up period 97 days: range 8 to 167 days). Percutaneous DC shocks up to 250 J proved to be a safe technique for closed-chest ablation of conduction tissue in dogs. These might be of value for patients with arrhythmias requiring electrical ablation of accessory pathways or foci situated in various sites in the heart.

Key Words: Closed-chest ablation of conduction • intracardiac direct-current shocks in dogs • electric injury of myocardium • arrhythmias


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 Am. J. Physiol. Heart Circ. Physiol.Home page
J. A. Barrabes, D. Garcia-Dorado, M. A. Gonzalez, M. Ruiz-Meana, J. Solares, Y. Puigfel, and J. Soler-Soler
Regional expansion during myocardial ischemia predicts ventricular fibrillation and coronary reocclusion
Am J Physiol Heart Circ Physiol, May 1, 1998; 274(5): H1767 - H1775.
[Abstract] [Full Text] [PDF]


Disclaimer:
Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.