Skip Navigation

European Heart Journal 2003 24(2):133-135; doi:10.1016/S0195-668X(02)00609-7
Copyright © 2003 by the European Society of Cardiology.
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Related articles in EHJ
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Zarco, P
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Zarco, P
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Editorial

Aortic stenosis

P Zarco

Clinic Hospital San Carlos, Madrid, Spain

The first 10% of the full text of this article appears below.

See doi:10.1016/S1095-668X(02)00385-8for the article to which this editorial refers.

The decline in the incidence of rheumatic fever and the increasing proportion of the population over age of 65 have together led to a rise in the number of patients with aortic stenosis (AS), which is now a common valvular lesion. The formerly termed senile and now age-related degenerative or dystrophic calcific AS is the most common cause of AS in adults and the most frequent reason for aortic valve replacement in patients with AS.

Calcific AS was described by Mönckeberg in 1904.1 The process observed in elderly individuals without any history of rheumatic fever in whom the mitral valve is normal, suggests a degenerative process. Calcification occurs in the bases of the cusp in the depth of the sinuses of . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?

Related articles in EHJ:

Lipoprotein(a), Chlamydia pneumoniae, leptin and tissue plasminogen activator as risk markers for valvular aortic stenosis
C.A Glader, L.S Birgander, S Söderberg, H.P Ildgruben, P Saikku, A Waldenström, and G.H Dahlén
EHJ 2003 24: 198-208. [Abstract] [Full Text]  



This article has been cited by other articles:


Home page
LupusHome page
A N Kiani, E K Fishman, and M Petri
Aortic valve calcification in systemic lupus erythematosus
Lupus, December 1, 2006; 15(12): 873 - 876.
[Abstract] [PDF]