Skip Navigation

European Heart Journal 2004 25(14):1185-1186; doi:10.1016/j.ehj.2004.04.014
Copyright © 2004 by the European Society of Cardiology.
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Related articles in EHJ
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (4)
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Goette, A.
Right arrow Articles by Röcken, C.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Goette, A.
Right arrow Articles by Röcken, C.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Editorial

Atrial amyloidosis and atrial fibrillation: a gender-dependent "arrhythmogenic substrate"?

Andreas Goettea,* and Christoph Röckenb

a Division of Cardiology, Otto-von-Guericke-University Hospital, Magdeburg, Germany
b Department of Pathology, Otto-von-Guericke-University Hospital, Magdeburg, Germany

* Correspondence to: A. Goette, Division of Cardiology, Otto-von-Guericke-University Hospital, Leipziger Str. 44, 39120 Magdeburg, Germany.
E-mail address: andreas.goette@medizin.uni-magdeburg.de

The first 10% of the full text of this article appears below.

This editorial refers to "Amyloid deposition as a cause of atrial remodelling in persistent valvular atrial fibrillation"1 by O. Leone et al. on page 1237

Atrial fibrillation (AF) is known to cause significant changes in atrial tissue architecture and electrophysiology.1,2 It has become clear over recent years that pre-existing alterations, such as autonomic dysbalance, degenerative tissue changes and fibrosis, can provide an electrophysiological and morphological substrate, which increases the likelihood of AF. In particular, alterations of the interstitial matrix in atrial tissue seem to be significant contributory factors.1 Increased amounts of fibrous tissue in fibrillating human atria were reported as early as 30 years ago3 and it is known to impair cell-to-cell . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?

Related articles in EHJ:

Amyloid deposition as a cause of atrial remodelling in persistent valvular atrial fibrillation
O Leone, G Boriani, B Chiappini, D Pacini, G Cenacchi, S Martin Suarez, C Rapezzi, M.L Bacchi Reggiani, and G Marinelli
EHJ 2004 25: 1237-1241. [Abstract] [Full Text]  



This article has been cited by other articles:


Home page
 CirculationHome page
R. H. Falk
Diagnosis and Management of the Cardiac Amyloidoses
Circulation, September 27, 2005; 112(13): 2047 - 2060.
[Full Text] [PDF]