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European Heart Journal 2004 25(14):1185-1186; doi:10.1016/j.ehj.2004.04.014
Copyright © 2004 by the European Society of Cardiology.
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Editorial

Atrial amyloidosis and atrial fibrillation: a gender-dependent "arrhythmogenic substrate"?

Andreas Goettea,* and Christoph Röckenb

a Division of Cardiology, Otto-von-Guericke-University Hospital, Magdeburg, Germany
b Department of Pathology, Otto-von-Guericke-University Hospital, Magdeburg, Germany

* Correspondence to: A. Goette, Division of Cardiology, Otto-von-Guericke-University Hospital, Leipziger Str. 44, 39120 Magdeburg, Germany.
E-mail address: andreas.goette@medizin.uni-magdeburg.de

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This editorial refers to "Amyloid deposition as a cause of atrial remodelling in persistent valvular atrial fibrillation"1 by O. Leone et al. on page 1237

Atrial fibrillation (AF) is known to cause significant changes in atrial tissue architecture and electrophysiology.1,2 It has become clear over recent years that pre-existing alterations, such as autonomic dysbalance, degenerative tissue changes and fibrosis, can provide an electrophysiological and morphological substrate, which increases the likelihood of AF. In particular, alterations of the interstitial matrix in atrial tissue seem to be significant contributory factors.1 Increased amounts of fibrous tissue in fibrillating human atria were reported as early as 30 years ago3 and it is known to impair cell-to-cell . . . [Full Text of this Article]


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Related articles in EHJ:

Amyloid deposition as a cause of atrial remodelling in persistent valvular atrial fibrillation
O Leone, G Boriani, B Chiappini, D Pacini, G Cenacchi, S Martin Suarez, C Rapezzi, M.L Bacchi Reggiani, and G Marinelli
EHJ 2004 25: 1237-1241. [Abstract] [FREE Full Text]  



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