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European Heart Journal Advance Access originally published online on November 30, 2004
European Heart Journal 2005 26(1):3-4; doi:10.1093/eurheartj/ehi041
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European Heart Journal (2005) 26, 3-4
© The European Society of Cardiology 2005

Venous and arterial thrombosis: a continuous spectrum of the same disease?

Carlos Jerjes-Sanchez*

Emergency Care Department, Hospital de Enfermedades Cardiovasculares y del Torax, IMSS, Monterrey, Mexico

online publish-ahead-of-print 30 November 2004 .

* Av. San Angel 130, Depto 5, Col. Valle de San Angel, CP 66290, Garza Garcia, NL, Mexico. Tel/fax: +52 81 83031641. E-mail address: jerjes@infosel.net.mx

The first 10% of the full text of this article appears below.

Over 150 years ago, Virchow1 first postulated that a triad of conditions predispose to thrombus formation, these three factors being abnormalities in the vessel wall, blood flow, and the coagulability of blood. The first two components of Rudolf Virchow's triad were based on his meticulous necropsy observations; the third reflects Virchow's remarkable insight that is only today, 150 years later, being substantiated at a molecular level. While Virchow was referring to venous thrombosis, the same processes have been applied to arterial thrombosis. A contemporary viewpoint of Virchow's triad should consider: inflammation, endothelial dysfunction, and atherosclerosis (i.e. abnormal vessel wall), abnormalities of haemorheology and turbulence at bifurcations and stenotic regions (i.e. abnormal blood flow), and finally, abnormalities in platelet function, coagulation, fibrinolysis, and metabolic . . . [Full Text of this Article]

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