Skip Navigation


European Heart Journal Advance Access originally published online on June 16, 2005
European Heart Journal 2005 26(16):1579-1581; doi:10.1093/eurheartj/ehi374
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow All Versions of this Article:
26/16/1579    most recent
ehi374v1
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Related articles in EHJ
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (1)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Toth, P. P.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Toth, P. P.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The European Society of Cardiology 2005. All rights reserved. For Permissions, please e-mail: journals.permissions@oupjournals.org

Adiponectin and high-density lipoprotein: a metabolic association through thick and thin

Peter P. Toth*

Sterling Rock Falls Clinic, 101 East Miller Road, Sterling, IL, USA

* Corresponding author. Tel: +1 815 632 5093; fax: +1 815 626 5947. E-mail address: peter.toth@srfc.com

This editorial refers to ‘Adiponectin, risk of coronary heart disease and correlation with cardiovascular risk markers’{dagger} by D. Rothenbacher et al., on page 1640

The first 150 words of the full text of this article appear below.

Obesity is highly prevalent. The increased availability of food, mechanization, lifestyle alterations, and other factors have made it easier for individuals to store rather than combust energy. Epidemiologic investigation has repeatedly demonstrated that obesity increases the risk for developing atherosclerotic disease and its clinical sequelae such as myocardial infarction (MI), ischaemic cerebrovascular accident, sudden death, and peripheral vascular disease. Obesity can directly and indirectly induce a large number of metabolic changes ultimately resulting in a cluster of risk factors collectively referred to as the ‘metabolic syndrome’. The metabolic syndrome is characterized by hypertension, dyslipidaemia (increased serum levels of small, dense LDL particles and triglycerides, and reduced HDL), insulin resistance with progressive deterioration in glucose tolerance, a pro-oxidative and pro-thrombotic state, and increased systemic inflammatory tone. The metabolic syndrome significantly augments risk for developing cardiovascular disease1 and diabetes mellitus and has become epidemic in the United States, Europe, and parts of . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?

Related articles in EHJ:

Adiponectin, risk of coronary heart disease and correlations with cardiovascular risk markers
Dietrich Rothenbacher, Hermann Brenner, Winfried März, and Wolfgang Koenig
EHJ 2005 26: 1640-1646. [Abstract] [FREE Full Text]  



This article has been cited by other articles:


Home page
 J. Clin. Endocrinol. Metab.Home page
K. J. Mukamal, R. H. Mackey, L. H. Kuller, R. P. Tracy, R. A. Kronmal, M. A. Mittleman, and D. S. Siscovick
Alcohol Consumption and Lipoprotein Subclasses in Older Adults
J. Clin. Endocrinol. Metab., July 1, 2007; 92(7): 2559 - 2566.
[Abstract] [Full Text] [PDF]