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European Heart Journal Advance Access originally published online on December 20, 2004
European Heart Journal 2005 26(4):317-318; doi:10.1093/eurheartj/ehi087
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© The European Society of Cardiology 2004. All rights reserved. For Permissions, please e-mail: journals.permissions@oupjournals.org

Atherosclerosis, thrombosis, and inflammatory risk factors, from history and the laboratory to real life

John H. Zarifis*

Cardiology Department, Papageorgiou Hospital, Solomou 12, Sykies, Thessaloniki, 56626 Greece

* Corresponding author. Tel: +30 2310204785; fax: +30 2310247450.E-mail address: aathanasiado@tee.gr

This editorial refers to ‘Association of European population levels of thrombotic and inflammatory factors with risk of coronary heart disease: the MONICA Optional Haemostasis Study’{dagger} by J. Yarnell et al., on page 332

The first 10% of the full text of this article appears below.

Atherosclerosis has existed as a named entity for less than a century. It is, however, a pathological process of very considerable antiquity; changes consistent with atherosclerosis have been found in the 18th dynasty of the pharaohs of Egypt. Despite this long history,1 our understanding of many of the fundamental aspects of the genesis and progression of atherosclerosis is still far from complete. The mechanism by which atherosclerotic lesions form and expand has puzzled scientists for 150 years. Rudolf Virchow proposed in 1856 that atherosclerosis was caused when plasma components (including lipids) elicited an inflammatory response in the arterial wall. With the advent of molecular medicine, it became possible to formulate . . . [Full Text of this Article]


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Related articles in EHJ:

Association of European population levels of thrombotic and inflammatory factors with risk of coronary heart disease: the MONICA Optional Haemostasis Study{dagger}
John Yarnell, Evelyn McCrum, Ann Rumley, Christopher Patterson, Veikko Salomaa, Gordon Lowe, Alun Evans, and on behalf of the MONICA Optional Haemostasis Study Investigators
EHJ 2005 26: 332-342. [Abstract] [FREE Full Text]