Skip Navigation


European Heart Journal Advance Access originally published online on March 11, 2005
European Heart Journal 2005 26(9):861-862; doi:10.1093/eurheartj/ehi220
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow All Versions of this Article:
26/9/861    most recent
ehi220v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Related articles in EHJ
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Goetze, J. P.
Right arrow Articles by Videbaek, R.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Goetze, J. P.
Right arrow Articles by Videbaek, R.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The European Society of Cardiology 2005. All rights reserved. For Permissions, please e-mail: journals.permissions@oupjournals.org

More hormones spilt in heart failure: linking renal sympathetic activation to clinical outcome

Jens Peter Goetze1,* and Regitze Videbaek2

1Department of Clinical Biochemistry, Rigshospitalet, University of Copenhagen, 9 Blegdamsvej, DK-2100 Copenhagen, Denmark
2Cardiac Catheterization Laboratory, Rigshospitalet, University of Copenhagen, 9 Blegdamsvej, DK-2100 Copenhagen, Denmark

* Corresponding author. Tel: +45 3545 5509; fax: +45 3545 4640. E-mail address: jpg@dadlnet.dk

This editorial refers to ‘Long-term outcome in relation to renal sympathetic activity in patients with chronic heart failure’{dagger} by M. Petersson et al., on page 906

The first 10% of the full text of this article appears below.

‘Neurohumoral activation’ is a commonly used term in papers dealing with heart failure. Although the term is short, the process is vast and complex: perhaps as complex as it gets. As heart failure affects almost all organs through reduced blood supply and compensatory regulation of auto-, para-, neuro-, and endocrine mechanisms, it seems overwhelming to rank the individual effect and regulation of one bioactive substance to the effects and regulations of others. Which substance affects which system and in what order through the course of disease progression? To make matters more complex, new substances are still being added to the long list of hormones, despite the fact that it has . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?

Related articles in EHJ:

Long-term outcome in relation to renal sympathetic activity in patients with chronic heart failure
Magnus Petersson, Peter Friberg, Graeme Eisenhofer, Gavin Lambert, and Bengt Rundqvist
EHJ 2005 26: 906-913. [Abstract] [Full Text]