Skip Navigation


European Heart Journal Advance Access originally published online on July 21, 2006
European Heart Journal 2006 27(16):1889-1890; doi:10.1093/eurheartj/ehl126
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow All Versions of this Article:
27/16/1889    most recent
ehl126v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Related articles in EHJ
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (2)
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Burke, A. P.
Right arrow Articles by Virmani, R.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Burke, A. P.
Right arrow Articles by Virmani, R.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The European Society of Cardiology 2006. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org

IVUS-VH: a predictor of plaque morphology?

Allen P. Burke, Michael Joner and Renu Virmani*

CVPath, International Registry of Pathology, 19 Firstfield Road, Gaithersburg, MD 20878, USA

* Corresponding author. Tel: +1 301 208 2490; fax: +1 301 208 3745. E-mail address: rvirmani@cvpath.org

This editorial refers to ‘Global characterization of coronary plaque rupture phenotype using three-vessel intravascular ultrasound radiofrequency data analysis’{dagger} by G.A. Rodriguez-Granillo et al., on page 1921

The first 150 words of the full text of this article appear below.

Imaging modalities that identify plaques vulnerable to rupture offer the possibility of preventing the most common substrate of coronary thrombosis. The incidence of progression of thin-cap atheroma, as identified at PCI, is largely unknown. Glaser et al.1 have demonstrated that at 1-year follow-up of a cohort of patients undergoing PCI, at least 6% develop progression of disease requiring additional PCI for another target lesion. The major predictors of plaque progression were multivessel disease, prior PCI, and age less than 65 years. Goldstein et al.2 demonstrated that 17% of acute MI patients with multiple complex angiographic lesions undergo PCI of non-culprit lesions within 1 year. These data underscore the need for identifying those plaques in high-risk patients who will benefit from prophylactic intervention. The hope is that newer imaging modalities, such as IVUS-virtual histology (VH) will precisely identify those lesions.

Rodriguez-Granillo et al.3 describe IVUS-VH in a prospective study of 40 . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?

Related articles in EHJ:

Global characterization of coronary plaque rupture phenotype using three-vessel intravascular ultrasound radiofrequency data analysis
Gastón A. Rodriguez-Granillo, Héctor M. García-García, Marco Valgimigli, Sophia Vaina, Carlos van Mieghem, Robert J. van Geuns, Maarten van der Ent, Evelyn Regar, Peter de Jaegere, Willem van der Giessen, Pim de Feyter, and Patrick W. Serruys
EHJ 2006 27: 1921-1927. [Abstract] [Full Text]  



This article has been cited by other articles:


Home page
 Eur Heart JHome page
S. K. Mehta, J. R. McCrary, A. D. Frutkin, W. J.S. Dolla, and S. P. Marso
Intravascular ultrasound radiofrequency analysis of coronary atherosclerosis: an emerging technology for the assessment of vulnerable plaque
Eur. Heart J., June 1, 2007; 28(11): 1283 - 1288.
[Abstract] [Full Text] [PDF]