Skip Navigation


European Heart Journal Advance Access originally published online on October 4, 2006
European Heart Journal 2006 27(21):2485-2486; doi:10.1093/eurheartj/ehl284
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Supplementary Data
Right arrow All Versions of this Article:
27/21/2485    most recent
ehl284v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Related articles in EHJ
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (1)
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Caligiuri, G.
Right arrow Articles by Nicoletti, A.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Caligiuri, G.
Right arrow Articles by Nicoletti, A.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The European Society of Cardiology 2006. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org

Lymphocyte responses in acute coronary syndromes: lack of regulation spawns deviant behaviour

Giuseppina Caligiuri* and Antonino Nicoletti

Centre de Recherche des Cordeliers, Université Pierre et Marie Curie-Paris6, Inserm UMR S 681, 15 rue de L'Ecole de Medecine, Paris F-75006, France

* Corresponding author. Tel: +33 155428269; fax: +33 155428262. E-mail address: giuseppina.caligiuri@umrs681.jussieu.fr

This editorial refers to ‘Altered status of CD4+CD25+ regulatory T cells in patients with acute coronary syndromes’{dagger} by A. Mor et al., on page 2530

The first 10% of the full text of this article appears below.

The inflammatory and immune systems are abnormally activated during the acute phases of coronary atherosclerotic disease.1,2 In particular, an unusual population of effector cells, lacking costimulatory molecule CD28, is transiently expanded in parallel with the occurrence of the waxing phases of atherosclerotic coronary disease.3

The origin of the lymphocyte response in acute coronary syndromes has been a matter of debate in the last decade. Questions still overcome the answers. It has been demonstrated that the T-cell response can be directed to restricted antigens within the . . . [Full Text of this Article]

Supplementary material


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?

Related articles in EHJ:

Altered status of CD4+CD25+ regulatory T cells in patients with acute coronary syndromes
Adi Mor, Galia Luboshits, David Planer, Gad Keren, and Jacob George
EHJ 2006 27: 2530-2537. [Abstract] [Full Text]  



This article has been cited by other articles:


Home page
 J Am Coll CardiolHome page
E. Groyer, A. Nicoletti, H. Ait-Oufella, J. Khallou-Laschet, A. Varthaman, A.-T. Gaston, O. Thaunat, S. V. Kaveri, R. Blatny, H. Stockinger, et al.
Atheroprotective Effect of CD31 Receptor Globulin Through Enrichment of Circulating Regulatory T-Cells
J. Am. Coll. Cardiol., July 24, 2007; 50(4): 344 - 350.
[Abstract] [Full Text] [PDF]