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European Heart Journal Advance Access originally published online on October 4, 2006
European Heart Journal 2006 27(21):2485-2486; doi:10.1093/eurheartj/ehl284
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© The European Society of Cardiology 2006. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org

Lymphocyte responses in acute coronary syndromes: lack of regulation spawns deviant behaviour

Giuseppina Caligiuri* and Antonino Nicoletti

Centre de Recherche des Cordeliers, Université Pierre et Marie Curie-Paris6, Inserm UMR S 681, 15 rue de L'Ecole de Medecine, Paris F-75006, France

* Corresponding author. Tel: +33 155428269; fax: +33 155428262. E-mail address: giuseppina.caligiuri@umrs681.jussieu.fr

This editorial refers to ‘Altered status of CD4+CD25+ regulatory T cells in patients with acute coronary syndromes’{dagger} by A. Mor et al., on page 2530

The first 10% of the full text of this article appears below.

The inflammatory and immune systems are abnormally activated during the acute phases of coronary atherosclerotic disease.1,2 In particular, an unusual population of effector cells, lacking costimulatory molecule CD28, is transiently expanded in parallel with the occurrence of the waxing phases of atherosclerotic coronary disease.3

The origin of the lymphocyte response in acute coronary syndromes has been a matter of debate in the last decade. Questions still overcome the answers. It has been demonstrated that the T-cell response can be directed to restricted antigens within the . . . [Full Text of this Article]

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Related articles in EHJ:

Altered status of CD4+CD25+ regulatory T cells in patients with acute coronary syndromes
Adi Mor, Galia Luboshits, David Planer, Gad Keren, and Jacob George
EHJ 2006 27: 2530-2537. [Abstract] [FREE Full Text]  



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