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European Heart Journal Advance Access originally published online on March 11, 2008
European Heart Journal 2008 29(7):846-848; doi:10.1093/eurheartj/ehn055
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please email: journals.permissions@oxfordjournals.org

Evolving concepts of left ventricular hypertrophy

Valérie Tikhonoff and Edoardo Casiglia*

Department of Clinical and Experimental Medicine, University of Padova, Via Giustiniani, 2, 35128 Padova, Italy

* Corresponding author. Email: edoardo.casiglia@unipd.it

This editorial refers to ‘Characterization of the GNAQ promoter and association of increased Gq expression with cardiac hypertrophy in humans’{dagger} by U.H. Frey et al., on page 888

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The opinions expressed in this article are not necessarily those of the Editors of the European Heart Journal or of the European Society of Cardiology.

The first 10% of the full text of this article appears below.

Adaptative left ventricular hypertrophy mainly derives from pressure or volume overload. Nevertheless, in the general population, about one-fifth of normotensives develop left ventricular hypertrophy despite a normal pressure load, while more than one-third of hypertensives do not develop it in response to pressure overload (Table 1). It is unclear why some subjects become hypertrophic while others do not. Hypoxic or ischaemic myocyte loss could account for a limited number of such cases. The natural history of left ventricular hypertrophy is also very . . . [Full Text of this Article]


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Related articles in EHJ:

Characterization of the GNAQ promoter and association of increased Gq expression with cardiac hypertrophy in humans
Ulrich H. Frey, Wolfgang Lieb, Jeanette Erdmann, Danai Savidou, Gerd Heusch, Kirsten Leineweber, Heinz Jakob, Hans-Werner Hense, Hannelore Löwel, Norbert H. Brockmeyer, Heribert Schunkert, and Winfried Siffert
EHJ 2008 29: 888-897. [Abstract] [FREE Full Text]