Skip Navigation


European Heart Journal Advance Access originally published online on December 11, 2008
European Heart Journal 2009 30(2):127-128; doi:10.1093/eurheartj/ehn555
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow All Versions of this Article:
30/2/127    most recent
ehn555v1
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Related articles in EHJ
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Katus, H.A.
Right arrow Articles by Thygesen, K.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Katus, H.A.
Right arrow Articles by Thygesen, K.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please email: journals.permissions@oxfordjournals.org

Higher sensitivity troponin assays: Quo vadis?

H.A. Katus1, E. Giannitsis1, A.S. Jaffe2 and K. Thygesen3,*

1 Universitätsklinik Heidelberg, Heidelberg, Germany
2 Mayo Clinic, Rochester, Minnesota, USA
3 Department of Medicine and Cardiology A, Aarhus University Hospital, Tage Hansens Gade 2, Aarhus DK-8000, Denmark

* Corresponding author. Tel: +45 89497614, Fax: +45 89497619, Email: Kristian.Thygesen@as.aaa.dk; or kthygesen@oncable.dk

This editorial refers to ‘Detection of acute changes in circulating troponin in the setting of transient stress test-induced myocardial ischaemia using an ultrasensitive assay: results from TIMI 35’{dagger} by M.S. Sabatine et al. on page 162

Footnotes

The opinions expressed in this article are not necessarily those of the Editors of the European Heart Journal or of the European Society of Cardiology.

The first 10% of the full text of this article appears below.

Central to the universal definition of myocardial infarction (MI) is the recommendation that cardiac troponin (cTn; I or T) is the biomarker of choice for detection of myocardial necrosis because of its high clinical sensitivity and almost absolute myocardial tissue specificity.1 The guidelines are based on the assays presently available for clinical use. However, predicated on data suggesting that there is additional information below the levels that are presently being measured, manufacturers have begun to develop much more sensitive cTn assays that allow measurement of concentrations in the range of . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?

Related articles in EHJ:

Detection of acute changes in circulating troponin in the setting of transient stress test-induced myocardial ischaemia using an ultrasensitive assay: results from TIMI 35
Marc S. Sabatine, David A. Morrow, James A. de Lemos, Petr Jarolim, and Eugene Braunwald
EHJ 2009 30: 162-169. [Abstract] [Full Text]