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European Heart Journal Advance Access originally published online on March 4, 2009
European Heart Journal 2009 30(7):755-756; doi:10.1093/eurheartj/ehp092
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2009. For permissions please email: journals.permissions@oxfordjournals.org

Whose heart will get broken? Troponin testing and future heart failure

Roland R.J. van Kimmenade1,2 and James L. Januzzi, Jr2,*

1 Department of Cardiology, University Hospital Maastricht, The Netherlands
2 Division of Cardiology, Massachusetts General Hospital, Boston, MA 02114, USA

* Corresponding author. Tel: +1 617 726 3443, Fax: +1 617 643 1620, Email: jjanuzzi@partners.org

This editorial refers to ‘Cardiac troponin-I and risk of heart failure: a community-based cohort study’{dagger}, by J. Sundström et al., on page 773

The first 10% of the full text of this article appears below.

After the introduction of troponin T (cTnT) and later troponin I (cTnI) as useful markers of myocardial necrosis,1 their testing has quickly redefined clinical cardiology. Indeed, the importance of troponin (cTn) for the diagnosis of myocardial injury has recently been reaffirmed in the ‘Universal Definition of Myocardial Infarction’: ‘if biomarkers have been measured ... and are normal, the determinations of these take precedence over ... imaging criteria’.2 This well illustrates that despite all technological developments in imaging techniques over the last two decades, cTn testing remains the gold standard for the identification of acute myocardial injury.

There is, however, a potential weakness in cTn testing that is widely acknowledged: notwithstanding the superior . . . [Full Text of this Article]


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Cardiac troponin-I and risk of heart failure: a community-based cohort study
Johan Sundström, Erik Ingelsson, Lars Berglund, Björn Zethelius, Lars Lind, Per Venge, and Johan Ärnlöv
EHJ 2009 30: 773-781. [Abstract] [Full Text]  



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