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European Heart Journal Advance Access originally published online on April 9, 2009
European Heart Journal 2009 30(9):1035-1037; doi:10.1093/eurheartj/ehp154
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2009. For permissions please email: journals.permissions@oxfordjournals.org
The online version of this article has been published under an open access model. Users are entitled to use, reproduce, disseminate, or display the open access version of this article for non-commercial purposes provided that the original authorship is properly and fully attributed; the Journal, Learned Society and Oxford University Press are attributed as the original place of publication with correct citation details given; if an article is subsequently reproduced or disseminated not in its entirety but only in part or as a derivative work this must be clearly indicated. For commercial re-use, please contact journals.permissions@oxfordjournals.org

Atrial fibrillation complicating acute myocardial infarction: how should it be interpreted and how should it be treated and prevented?

Riccardo Cappato*

Arrhythmia and Electrophysiology Department, Policlinico San Donato IRCCS, Milan, Italy

* Corresponding author. Tel: +39 02 5277 4337, Fax: +39 02 5560 3125, Email: rcappato@libero.it

This editorial refers to ‘Atrial fibrillation in acute myocardial infarction: a systematic review of the incidence, clinical features and prognostic implications’{dagger}, by J. Schmitt et al., on page 1038

The first 150 words of the full text of this article appear below.

Atrial fibrillation (AF) has been reported to complicate the course of acute myocardial infarction (AMI) in ~6–21% of hospitalized patients.1 Possible precipitating factors of AF in this setting include atrial ischaemia or infarction, right ventricular infarction, pericardial inflammation, acute hypoxia or hypokalaemia, and haemodynamic impairment secondary to left ventricular (LV) dysfunction.2–4 Endogenous or exogenous catecholamines may also precipitate AF. These factors can be found alone or in combination, and may superimpose on predisposing diseases affecting cardiac anatomy and physiology, such as previous cardiomyopathy, valvular impairment, or chronic lung disease. Finally, AF in the setting of AMI has been reported to be associated with ageing, severely impaired LV function, presence of mitral regurgitation, or frequent ventricular arrhythmias plus right bundle branch block, and presence of left bundle branch block. Although most of these factors are claimed to be causative of AF, the intimate relationship between their presence and AF occurrence is . . . [Full Text of this Article]

How should AF during AMI be interpreted?

How should AF during AMI be treated and prevented?

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Related articles in EHJ:

Atrial fibrillation in acute myocardial infarction: a systematic review of the incidence, clinical features and prognostic implications
Joern Schmitt, Gabor Duray, Bernard J. Gersh, and Stefan H. Hohnloser
EHJ 2009 30: 1038-1045. [Abstract] [FREE Full Text]