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European Heart Journal 2000 21(14):1113-1115; doi:10.1053/euhj.1999.2024
Copyright © 2000 by the European Society of Cardiology.
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Hotline Editorial

The VA HDL Intervention Trial: clinical implications

H.B. Rubinsa, D. Collinsb and S.J. Robinsc

a Center for Chronic Disease Outcomes Research, Veterans Affairs Medical Center, Minneapolis, Minnesota, U.S.A.
b Department of Veterans Affairs Cooperative Studies Program Coordinating Center, West Haven, Connecticut, U.S.A.
c Department of Medicine, Boston University School of Medicine, Boston, Massachusetts, U.S.A.

References

  1. Rubins HB, Robins SJ, Collins D. Distribution of lipids in 8,500 men with coronary artery disease. Am J Cardiol. 1995;75:1196–1201[CrossRef][Web of Science][Medline]
  2. Goldbourt U, Yaari S, Medalie JH. Isolated low HDL cholesterol as a risk factor for coronary heart disease mortality. A 21-year follow-up of 8000 men. Arterioscler Thromb Vasc Biol. 1997;17:107–113[Abstract/Free Full Text]
  3. Rubins HB, Robins SJ, Iwane MK. Rationale and design of the Department of Veterans Affairs High-Density Lipoprotein Cholesterol Intervention Trial (HIT) for secondary prevention of coronary artery disease in men with low high-density lipoprotein cholesterol an desirable low-density lipoprotein cholesterol. Am J Cardiol. 1993;71:45–52[CrossRef][Web of Science][Medline]
  4. Rubins HB, Robins SJ, Collins D. The Veterans Affairs High-Density Lipoprotein Intervention Trial: Baseline characteristics of normocholesterolemic men with coronary artery disease and low levels of high-density lipoprotein cholesterol. Am J Cardiol. 1996;78:572–575[CrossRef][Web of Science][Medline]
  5. Rubins HB, Robins SJ, Collins D. Gemfibrozil for the secondary prevention of coronary heart disease in men with low levels of high-density lipoprotein cholesterol. N Engl J Med. 1999;341:410–418[Abstract/Free Full Text]
  6. Sacks FM, Pfeffer MA, Moye LA. The effect of pravastatin on coronary events after myocardial infarction in patients with average cholesterol levels. N Engl J Med. 1996;335:1001–1009[Abstract/Free Full Text]
  7. N Engl J Med. 1998;339:1349–1357[Abstract/Free Full Text]
  8. Wilson PWF, Kannel WB, Silbershatz H, D'Agostino RB. Clustering of metabolic factors and coronary heart disease. Arch Intern Med. 1999;159:1104–1109[Abstract/Free Full Text]
  9. Grundy SM. Small LDL, atherogenic dyslipidemia, and the metabolic syndrome. Circulation. 1997;95:1–4[Free Full Text]
  10. Tenkanen L, Manttari M, Manninen V. Some coronary risk factors related to the insulin resistance syndrome and treatment with Gemfibrozil. Experience from the Helsinki Heart Study. Circulation. 1995;92:1779–1785[Abstract/Free Full Text]
  11. Schonfeld G. The effects of fibrates on lipoprotein and hemostatic coronary risk factors. Atherosclerosis. 1994;111:161–174[CrossRef][Web of Science][Medline]
  12. Staels B, Dallongeville J, Auwerx J, Schoonjans K, Leitersdorf E, Fruchart J-C. Mechanism of action of fibrates on lipid and lipoprotein metabolism. Circulation. 1998;98:2088–2093[Abstract/Free Full Text]
  13. DiPalma JR, Thayer WS. Use of niacin as a drug. Annu Rev Nutr. 1991;11:169–187[CrossRef][Web of Science][Medline]
  14. Goldbourt U, Behar S, Reiss-Reicher H. Rationale and design of a secondary prevention trial of increasing serum high-density lipoprotein cholesterol and reducing triglycerides in patients with clinically manifest atherosclerotic heart disease (the Bezafibrate Infarction Prevention Trial). Am J Cardiol. 1993;71:909–915[CrossRef][Web of Science][Medline]
  15. Grundy SM, Balady GJ, Criqui MH. When to start cholesterol-lowering therapy in patients with coronary heart disease. A statement for healthcare professionals from the American Heart Association Task Force on risk reduction. Circulation. 1997;95:1683–1685[Free Full Text]

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