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European Heart Journal 1999 20(24):1843; doi:10.1053/euhj.1999.1718
Copyright © 1999 by the European Society of Cardiology.
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Letters to the Editor

Sporadic cases of dilated cardiomyopathies associated with atrioventricular conduction defects are not linked to mutation within the connexins 40 and 43 genes

J.-A. Haefliger, J.J. Goy and G. Waeber

University Hospital, Lausanne, Switzerland

References

  1. Manolio TA, Baughman KL, Rodeheffer R. Prevalence and etiology of idiopathic dilated cardiomyopathy (summary of a National Heart, Lung, and Blood Institute workshop). Am J Cardiol. 1992;1:1458–1466
  2. Muntoni F, Cau M, Ganau A. Deteletion of the dystrophin muscle-promoter region associated with X-linked dilated cardiomyopathy. N Engl J Med. 1993;329:921–925[Free Full Text]
  3. Muntoni F, Di Lenarda A, Porcu M. Dystrophin gene abnormalities in two patients with idopathic dilated cardiomyopathy. Heart. 1997;78:608–612[Abstract/Free Full Text]
  4. Wynne J, Braunwald E. The cardiomyopathies and myocarditis: toxic, chemical and physical damage to the heart. Braunwald E. Heart Disease. Philadelphia: W. B. Saunders; 1992. p. 1394–1450
  5. Michels VV, Moll PP, Miller FA. The frequency of familial dilated cardiomyopathy in a series of patients with idiopathic dilated cardiomyopathy. N Engl J Med. 1992;326:77–82[Abstract]
  6. Kass S, MacRae C, Graber HL. A gene defect that causes conduction system disease and dilated cardiomyopathy maps to chromosome 1p1-1q1. Nat Gen. 1994;7:546–551[CrossRef][Web of Science][Medline]
  7. Simon AM, Goodenough DA, Paul DL. Mice lacking connexin 40 have cardiac conduction abnormalities characteristic of atrioventricular block and bundle branch block. Cur Biol. 1998;8:295–298[CrossRef][Web of Science][Medline]
  8. Kirchhoff S, Nelles E, Hagendorf A, Krüger O, Traub O, Willecke K. Reduced cardiac conduction velocity and predisposition to arrhythmias in connexin 40-deficient mice. Cur Biol. 1998;8:299–302[CrossRef][Web of Science][Medline]
  9. Beyer EC, Kistler J, Paul DL, Goodenough DA. Antisera directed against connexin 43 peptides react with 43kD protein localized to gap junctions in myocardium and other tissues. J Cell Biol. 1989;108:595–605[Abstract/Free Full Text]
  10. Chen SC, Davis LM, Westphale EM, Beyer EC, Saffitz JE. Expression of multiple gap junction proteins in human fetal and infant hearts. Pediatr Res. 1994;36:561–566[Web of Science][Medline]
  11. Peters NS, Green CR, Poole-Wilson PA, Severs NJ. Reduced content of connexin 43 gap junctions in ventricular myocardium from hypertrophied and ischemic human hearts. Circulation. 1993;88:864–875[Abstract/Free Full Text]
  12. Haefliger JA, Castillo E, Waeber G. Hypertension increases connexin 43 in a tissue-specific manner. Circulation. 1997;95:1007–1014[Abstract/Free Full Text]
  13. Kanter HL, Saffitz JE, Beyer EC. Molecular cloning of two human cardiac gap junction proteins, connexin 40 and connexin 45. Mol Cell Cardiol. 1994;26:861–868[CrossRef][Web of Science][Medline]

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This Article
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