Skip Navigation


European Heart Journal Advance Access originally published online on November 30, 2004
European Heart Journal 2005 26(1):99; doi:10.1093/eurheartj/ehi029
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow All Versions of this Article:
26/1/99    most recent
ehi029v1
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Ostadal, P.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Ostadal, P.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Letter to the Editor

What is ‘reperfusion injury’?

Petr Ostadal

Department of Cardiology
2nd Faculty of Medicine
Charles University
University Hospital Motol
V Uvalu 84
150 18 - Prague 5
Czech Republic
E-mail address: ostadal@yahoo.com

The first 10% of the full text of this article appears below.

I read with great interest the article by Turschner et al.1 describing the echocardiographic observation of experimentally induced myocardial infarction and subsequent reperfusion in the pig model. They have demonstrated an increase in regional wall thickness in the reperfused infarct zone due to ‘oedema associated with reperfusion injury’.

. . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?