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European Heart Journal Advance Access published online on March 11, 2008
European Heart Journal, doi:10.1093/eurheartj/ehn055
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please email: journals.permissions@oxfordjournals.org

Evolving concepts about left ventricular hypertrophy

Valérie Tikhonoff and Edoardo Casiglia*

Department of Clinical and Experimental Medicine, University of Padova, Via Giustiniani, 2, 35128 Padova, Italy

* Corresponding author. Email: edoardo.casiglia@unipd.it

This editorial refers to ‘Characterization of the GNAQ promoter and association of increased Gq expression with cardiac hypertrophy in humans’ by U.H. Frey et al. doi:10.1093/eurheartj/ehm618

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Adaptative left ventricular hypertrophy mainly derives from pressure or volume overload. Nevertheless, in the general population, about one-fifth of normotensives develop left ventricular hypertrophy despite a normal pressure load, while more than one-third of hypertensives do not develop it in response to pressure overload (Table 1). It is unclear why some subjects become hypertrophic while others do not. Hypoxic or ischaemic myocyte loss could account for a limited number of such cases. The natural history of left ventricular hypertrophy is also very different, with some subjects developing heart failure and premature death and others who seem to be free of these prognostic effects1 (. . . [Full Text of this Article]


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Related articles in EHJ:

Characterization of the GNAQ promoter and association of increased Gq expression with cardiac hypertrophy in humans
Ulrich H. Frey, Wolfgang Lieb, Jeanette Erdmann, Danai Savidou, Gerd Heusch, Kirsten Leineweber, Heinz Jakob, Hans-Werner Hense, Hannelore Löwel, Norbert H. Brockmeyer, Heribert Schunkert, and Winfried Siffert
EHJ 2008 29: 888-897. [Abstract] [FREE Full Text]